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At one end of the spectrum is pure testosterone with no esters attached, such as testosterone suspension, where the testosterone esters are removed from the body and the testosterone is converted to estradiol (dihydrotestosterone) via a modified enzyme such as the aromatase enzyme. For pure testosterone, the concentration of testosterone in blood would normally be less than 0.1 ng/mL (which makes it a safe concentration for anabolic steroids). To find out more about the concentration of testosterone in your system, go to How Much Testosterone in Your Blood? And at the other end of the spectrum is synthetic testosterone, which contains testosterone esters linked to their conversion to estradiol, and is a form of testosterone not usually used in doping control in sports. Dissociation of the Testosterone Ester: Progesterone (Estroesterone) Testosterone and estradiol are chemically linked together, or conjugated. The enzyme aromatase removes esters from the testosterone esters, and makes estradiol. If you had a hormone test in the late 1940s, you'd have a high level of testosterone because the ester bonds were short. That's why when there's a chemical change of the ester bonds in a certain amount of testosterone, it usually means an increase in the concentration of testosterone in the blood. Since then, however, there have been a number of studies that have shown the exact opposite. A study of a group of women who were undergoing chemotherapy that involved estradiol (the female sex hormone) was published in the journal Blood by an Israeli researcher (Berman et al., 1993). That study involved injecting estrogen, but they did not increase the dose of aromatase or increase the number of follicles and/or the number of cells in a small artery or vein in one of the groups. So it looks like estradiol in cancer treatment is helping to lower testosterone in the blood. So, we have two different theories. One is that the ester bonds in the male or female testosterone and estrogen are linked by the enzyme. The other is that there are a variety of esters in the estrogens and it's not that the testosterone is converted to the estrogen. It's more that there are esters of testosterone in the testosterone esters, and this leads to a reduction in the concentration. The second scenario is in humans, and it might explain why there are not as many studies on testosterone and estradiol in cancer patients. As we know, Related Article:
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